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Acute Abdomen Updated 2026-04

Bowel Ischemia — CT Classification, SMA Embolus vs Thrombosis, and Mesenteric Ischemia

Mesenteric ischemia CT imaging: SMA embolus vs thrombosis, SMV thrombosis, NOMI, colonic watershed ischemia, strangulation, pneumatosis, and portal venous gas — urgent reporting guide.

Quick summary

Identify the etiology (embolus vs. thrombosis vs. venous vs. NOMI) — each has distinct CT appearance and management. SMA embolus spares the proximal jejunum; SMA thrombosis involves the entire territory from the origin. Pneumatosis + portal venous gas = transmural necrosis — call surgery immediately.

SMA Embolus vs Thrombosis

SMA embolus: Filling defect typically 3–10 cm from SMA origin (beyond origin at middle colic artery), often cardiac source (Afib, endocarditis); spares proximal jejunum.

SMA thrombosis: Occlusion at or near SMA origin, atherosclerotic disease, ischemia of entire SMA territory including proximal jejunum; longer segment ischemia; prior intestinal angina history.

Etiology and CT Findings

Etiology CT Findings Territory
SMA occlusion — embolus Filling defect in SMA 3–10 cm from origin; bowel wall thickening → thinning (necrosis); pneumatosis; portal venous gas; lack of bowel wall enhancement; mesenteric stranding Mid-jejunum to transverse colon; spares proximal jejunum and duodenum (SMA branches beyond origin occluded)
SMA occlusion — thrombosis Atherosclerotic calcification/plaque at SMA origin; near-complete or complete occlusion at origin; extensive small bowel and right colon ischemia; collateral vessels may be visible Entire SMA territory including proximal jejunum; duodenum may be involved; longer ischemic segment than embolus
SMV thrombosis Hyperdense SMV (acute thrombus); filling defect in SMV ± portal vein; mesenteric edema and ascites (venous congestion); bowel wall thickening (target sign/halo); hemorrhagic infarction more common than arterial; pneumatosis less common early Variable; often patchy; mesenteric venous distribution; may spare arterially supplied areas; hypercoagulable states, portal hypertension, pancreatitis
Non-occlusive mesenteric ischemia (NOMI) Patent mesenteric vessels; diffuse bowel wall thickening; poor bowel wall enhancement; mesenteric edema; no filling defect; SMA vasospasm may be visible on angiography Watershed areas most vulnerable: splenic flexure, sigmoid; diffuse distribution possible; ICU/low-flow states, dialysis, vasopressors
Colonic ischemia (watershed) Bowel wall thickening ± thumbprinting (submucosal edema); pericolonic stranding; watershed zones; typically no occlusion on CT; pneumatosis in severe cases Splenic flexure (Griffiths point) and rectosigmoid junction (Sudeck point) — watershed zones between SMA/IMA and IMA/internal iliac territories
Strangulation / closed-loop Closed loop morphology + wall thickening + loss of enhancement + mesenteric engorgement + ascites + pneumatosis; combination of arterial and venous compromise due to twisting Isolated closed loop; variable size; sigmoid or internal hernia most common; rapidly progressive to full-thickness necrosis

Critical finding: Pneumatosis intestinalis (intramural gas) + portal venous gas = grave prognosis — indicates advanced bowel necrosis with transmural infarction. Report immediately and obtain urgent surgical consultation. Distinguish pneumatosis from intraluminal gas: pneumatosis follows bowel wall contour, seen on multiple planes, often linear or bubbly in pattern.

References

Menke J. Diagnostic accuracy of MDCT for acute mesenteric ischemia: A systematic review. Eur Radiol. 2010;20(12):2805–14.

Radiopaedia — Bowel ischaemia


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